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manuals for sonyUsing research from 1980 onward, this book is a critical review of past and up-to-date research findings and concepts on the effects of noise on people; it focuses on the psychological and physiological affects of noise on hearing and performance. This text elucidates the interrelations of the acoustical, physiological, psychological, and sociological factors that are involved in making noise a problem to individuals and societies. Also discussed are hearing loss, speech communications, annoyance, and health effects criteria for the limitation of exposures to noise in living and work areas. It covers: physical characteristics of sound and noise; acoustical-sensorineural response characteristics of the ear; basic psychological sensations and perceptions that ensue from analysis of sound and noise by the auditory system; laboratory and real-life research on the impairments to hearing, speech communication, task performance, and mental and bodily health that occur from exposure to noise; and, physical measures which predict adverse effects on hearing, behavior, and health from exposure to noise.Your current browser may not support copying via this button. The 13-digit and 10-digit formats both work. Please try again. Used: GoodPages may include limited notes and highlighting. May not include supplemental or companion materials if applicable. Access codes may or may not work. Connecting readers since 1972. Customer service is our top priority.We'll e-mail you with an estimated delivery date as soon as we have more information. Your account will only be charged when we ship the item. It contains information of interest to government decision makers, architects, and engineers concerned with criteria, guidelines, and standards for acceptable and safe levels of sound and noise. Then you can start reading Kindle books on your smartphone, tablet, or computer - no Kindle device required. This is a most comprehensive handbook and it is difficult to point to any major omissions.http://gusanhightec.com/userData/board/ford-172-diesel-engine-manual.xml
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Full content visible, double tap to read brief content. Videos Help others learn more about this product by uploading a video. Upload video To calculate the overall star rating and percentage breakdown by star, we don’t use a simple average. Instead, our system considers things like how recent a review is and if the reviewer bought the item on Amazon. It also analyzes reviews to verify trustworthiness. Please try again later. Catherine Conroy 4.0 out of 5 stars Through my work I was inspired to learn as much as I could about hearing difficulties. This book was one of the first that I read on the subject, and I was thouroughly impressed. Not only did Kryter delve into the causes of loss, but also explored ways to prevent common problems. I reccomend this book to anyone who wants to learn about their hearing, and it is on our Must Read list at the office. Please choose a different delivery location or purchase from another seller.Please choose a different delivery location or purchase from another seller.Please try again. Please try your request again later. Then you can start reading Kindle books on your smartphone, tablet, or computer - no Kindle device required. Full content visible, double tap to read brief content. By continuing to browseFind out about Lean Library here Find out more and recommend Lean Library. Download PDFThis product could help you Lean Library can solve it Simply select your manager software from the list below and click on download.Simply select your manager software from the list below and click on download.For more information view the SAGE Journals Sharing page. Search Google ScholarSearch Google ScholarGoogle Scholar Moore, B C J, 1989 An Introduction to the Psychology of Hearing ( London: Academic Press ). Google Scholar Find out about Lean Library here Search Google ScholarBy continuing to browse. Auditory Sensations and Perceptions. Normal Hearing, Sociocusis, Nosocusis, and Hearing Loss from Industrial Noise.http://www.royal-pizza.eu/files/by-manual.xml Derivation of a General Theory and Procedure for Predicting Hearing Loss from Exposure to Sound. Speech Intelligibility in Quiet and in Noise. The Assessment of Hearing Handicap and Damage Risk from Noise. Mental and Psychomotor Task Performance in Noise. Jenna Clark, S. Adams, Allyson D. Dykstra, S. Moodie, M. Jog Psychology, Medicine Journal of communication disorders 2014 51 PDF Save Alert Research Feed. 1 2 3 4 5. Related Papers Abstract 128 Citations Related Papers Stay Connected With Semantic Scholar Sign Up What Is Semantic Scholar. Semantic Scholar is a free, AI-powered research tool for scientific literature, based at the Allen Institute for AI. Groups Discussions Quotes Ask the Author Using research from 1980 onward, this book is a critical review of past and up-to-date research findings and concepts on the effects of noise on people; it focuses on the psychological and physiological affects of noise on hearing and performance. This text elucidates the Using research from 1980 onward, this book is a critical review of past and up-to-date research findings and concepts on the effects of noise on people; it focuses on the psychological and physiological affects of noise on hearing and performance. Also discussed are hearing loss, speech communications, annoyance, and health effects criteria for the limitation of exposures to noise in living and work areas.To see what your friends thought of this book,This book is not yet featured on Listopia.There are no discussion topics on this book yet. San Diego: Academic Press, To learn more about how to request items watch this short online video. We will contact you if necessary. Please also be aware that you may see certain words or descriptions in this catalogue which reflect the author’s attitude or that of the period in which the item was created and may now be considered offensive.https://labroclub.ru/blog/enterasys-b2h124-48p-manual It contains information of interest to government decision makers, architects, and engineers concerned with criteria, guidelines, and standards for acceptable and safe levels of sound and noise. Kryter has successfully integrated masses of research in each chapter, and readers will be truly impressed with the organization, amount, and critical review of the information presented. This is a most comprehensive handbook and it is difficult to point to any major omissions.All Rights Reserved. Learn More. This article is an open-access article distributed under the terms and conditions of the Creative Commons Attribution license ( ). This article has been cited by other articles in PMC. Abstract Noise is defined as an unwanted sound or a combination of sounds that has adverse effects on health. These effects can manifest in the form of physiologic damage or psychological harm through a variety of mechanisms. Chronic noise exposure can cause permanent threshold shifts and loss of hearing in specific frequency ranges. Noise induced hearing loss (NIHL) is thought to be one of the major causes of preventable hearing loss. Approximately 10 million adults and 5.2 million children in the US are already suffering from irreversible noise induced hearing impairment and thirty million more are exposed to dangerous levels of noise each day. The mechanisms of NIHL have yet to be fully identified, but many studies have enhanced our understanding of this process. The role of oxidative stress in NIHL has been extensively studied. There is compelling data to suggest that this damage may be mitigated through the implementation of several strategies including anti-oxidant, anti-ICAM 1 Ab, and anti JNK intervention. The psychological effects of noise are usually not well characterized and often ignored. However, their effect can be equally devastating and may include hypertension, tachycardia, increased cortisol release and increased physiologic stress. Collectively, these effects can have severe adverse consequences on daily living and globally on economic production. This article will review the physiologic and psychologic consequences of noise and its effect on quality of life. Keywords: noise, oxidative stress, quality of life, noise and non-auditory health 1.?Introduction Noise is typically defined as an unwanted sound or a combination of sounds that may adversely affect people. Noise can manifest in the form of physiologic damage or psychological harm. The mechanism of physiological damage from noise has yet to be fully understood, but research has demonstrated a multitude of factors including increased oxidative stress, vascular changes, and mechanical trauma may be responsible, to name a few. Currently, the armed forces are facing severe disabilities secondary to noise. It has been shown that the top two disabilities now facing the American military are hearing loss and tinnitus. The US government is predicted to spend nearly 1.6 billion dollars this year in order to rehabilitate the men and women traumatized from the effects of noise (TATRC 2010). This results in bilateral, sensorineural hearing loss, often with a pathognomonic notch of decreased hearing on an audiogram at 4,000 Hz. The role of genetics has been a recent area of interest in uncovering the causative mechanisms of NIHL. There is anecdotal evidence suggesting a possible genetic role. They found a resistance to NIHL after prolonged noise exposure in mice when the wild type Ahl3 allele was present, demonstrating a genetic variation in susceptibility to NIHL. Hearing loss from otologic trauma, which is defined as a sudden change in hearing due to exposure of a sudden burst of sound, may be due to a different mechanism of action than NIHL, and should be thought of as a separate entity. Acoustic trauma can cause mechanical disruption of the cochlea and may result in permanent hearing loss. Noise induced hearing loss, on the other hand, is thought to be due to various other mechanisms. One leading theory that will be focused on in this article is the role of oxidative stress in chronic noise exposure. Exposure to noise has been demonstrated to cause an initial increase in cochlear blood flow. Research on oxidative stress has spawned subsequent attention toward the protective role of antioxidants in NIHL. The senior author has published multiple studies on the role of anti-oxidants in protection against NIHL. One of these studies evaluated the effects of resveratrol on NIHL. Resveratrol is a substance found in the skin of grapes and over 70 other fruits and plants. In this 2003 study by Seidman et al., Fischer rats were randomized into a treatment and control group. The treatment group were then given four weeks of post-stimulus treatment while the control group received only saline. The results from this study demonstrated a significant reduction of auditory threshold shifts in the treatment group compared to the controls. There was also a decrease in the loss of hair cells in the Organ of Corti in the treatment group compared to the control group. In this study, Fischer rats were randomized into a control group with saline and a treatment group where they received intravenous Anti-ICAM-1 Ab prior to a 72-hour period of 107 dB SPL noise. The groups then received another dose of Anti-ICAM-1 Ab or saline 24 hours after noise exposure. The rats treated with Anti-ICAM-1 Ab showed attenuated temporary threshold shifts (TTS) compared to the control group. There have been numerous studies on the role of oxidative stress in hearing loss and the ability of various anti-oxidants to attenuate the physiological damage. Another hypothesis looked at the role of glutamate in NIHL. Noise exposure is known to cause excessive excitatory synaptic activation of glutamate receptors and leads to glutamate excitotoxicity. This process ultimately results in neural swelling through water and calcium influx, which induces necrotic and apoptotic cell death in the spiral ganglion. Quantifying the non-auditory effects of noise may be difficult due to lack of strong scientific evidence lack statistical evidence, as there are often plausible alternative explanations for the results. These studies often have confounding variables, especially selection bias. There have been multiple studies that have investigated the relationship between noise, blood pressure, and myocardial damage. They found that there was a significant association between occupational and air-traffic noise exposure and hypertension. The evidence of noise exposure causing ischemic heart disease was inconclusive as there was no definitive relationship. Another study investigated the relationship of noise and blood pressure and a stress response included a quality of life survey to noise exposed subjects. They measured systolic and diastolic blood pressure, urine epinephrine, norepinephrine, and cortisol levels, as well as obtained results from the KINDL quality of life questionnaire. They found that there was an increase in blood pressure in the noisy communities. There was also a significant increase in urine epinephrine and norepinephrine levels, but not a significant change in cortisol levels. Noise can cause immediate or secondary extra-auditory effects. The number and duration of nighttime awakenings can quantify the immediate effect of noise on sleep. These psychological and physiological non-auditory effects of noise result in detrimental health consequences and a decreased quality of life. 4.?Conclusions This is just some of the evidence to highlight noise as an unwanted environmental pollutant that has global implications. In our industrialized society, a significant population is exposed to noise on a daily basis with its resultant health effects, and subsequent substantial economic burden. There has been a plethora of research on the mechanism of NIHL. The role of oxidative stress in NIHL has been extensively investigated and remains a probable cause of NIHL. This results in significant deterioration in quality of life in that it disrupts sleep, causes cognitive impairment, and has many non-auditory deleterious health effects. References and Notes 1. Public Health Service. Healthy People 2000: National Health Promotion and Disease Prevention Objectives. US Government Printing Office; Washington, DC, USA: 1990. Available online: (Site reviewed on 24 September 2007, accessed on 2 August 2010). 4. Smith A. The Fifteenth Most Serious Health Problem, The WHO Perspective. EPA; Washington, DC, USA: 1981. Criteria for a Recommended Standard: Occupational Noise Exposure Revised Criteria. NIOSH; Cincinnati, OI, USA: 1998. Chen TJ, Chen S-S, Wang D-C, Hsieh Y-L. Increased vulnerability of auditory system to noise exposure in mdx mice. Fairfield DA, Lomax M, Dootz G, Chen S, Galecki A, Benjamin I, Dolan D, Altschuler RA. Lurie M, Davis H, Hawkins J., Jr Acoustic trauma of the organ of Corti in the guinea pig. Lim DJ, Melnick W. Acoustic damage of the cochlea. Darrat I, Ahmad N, Seidman K, Seidman MD. Quirk WS, Seidman MD. Seidman M, Babu S, Tang W, Naem E, Quirk W. Effects of resveratrol on acoustic trauma. Seidman M, Tang W, Shirwany N, Bai U, Rubin C, Henig J, Quirk W. Anti-intercellular adhesion molecule-1 antibody's effect on noise damage. Samson J, Wiktorek-Smagur A, Politanski P, Rajkowska E, Pawlaczyk-Luszczynska M, Dudarewicz A, Sha H, Schacht J, Sliwinska-Kowalski M. Noise-induced time-dependent changes in oxidative stress in the mouse cochlea and attenuation by D-methionine. Bullinger R-S. Kindle Questionaire. Ravens-Sieberer U, Erhart M, Wetzel R, Bullinger M. Generic health-related quality-of-life assessment in children and adolescents: methodological considerations. Stevanovic D. Serbian KINDL questionnaire for quality of life assessments in healthy children and adolescents: reproducibility and construct validity. Stansfeld S, Matheson M. Noise pollution: Non-auditory effects on health. Hunt SM, McEwen J, McKenna SP. Reimer MA, Flemons WW. Ware JE, Jr, Sherbourne CD. Weaver TE, Laizner AM, Evans LK, Maislin G, Chugh DK, Lyon K, Smith PL, Schwartz AR, Redline S, Pack AI, Dinges DF. Ljung R, Sorqvist P, Hygge S. Effects of road traffic noise and irrelevant speech on children's reading and mathematical performance. Please try again.Please try your request again later. No Cost EMI availableSign up for free Using research from 1980 onward, this book is a critical review of past and up-to-date research findings and concepts on the effects of noise on people; it focuses on the psychological and physiological affects of noise on hearing and performance. Also discussed are hearing loss, speech communications, annoyance, and health effects criteria for the limitation of exposures to noise in living and work areas.Then you can start reading Kindle books on your smartphone, tablet, or computer - no Kindle device required. Get your Kindle here, or download a FREE Kindle Reading App.To calculate the overall star rating and percentage breakdown by star, we don’t use a simple average. It also analyses reviews to verify trustworthiness. Exposure to transport noise disturbs sleep in the laboratory, but not generally in field studies where adaptation occurs. Noise interferes in complex task performance, modifies social behaviour and causes annoyance. Studies of occupational and environmental noise exposure suggest an association with hypertension, whereas community studies show only weak relationships between noise and cardiovascular disease. Aircraft and road traffic noise exposure are associated with psychological symptoms but not with clinically defined psychiatric disorder. In both industrial studies and community studies, noise exposure is related to raised catecholamine secretion. In children, chronic aircraft noise exposure impairs reading comprehension and long-term memory and may be associated with raised blood pressure. Further research is needed examining coping strategies and the possible health consequences of adaptation to noise. Hearing impairments due to noise are a direct consequence of the effects of sound energy on the inner ear. However, the levels of environmental noise, as opposed to industrial noise, are much lower and effects on non-auditory health cannot be explained as a consequence of sound energy. If noise does cause ill-health other than hearing impairment, what might be the mechanism. It is generally believed that noise disturbs activities and communication, causing annoyance. In some cases, annoyance may lead to stress responses, then symptoms and possibly illness 3. Alternatively, noise may influence health directly and not through annoyance. The response to noise may depend on characteristics of the sound, including intensity, frequency, complexity of sound, duration and the meaning of the noise. Non-auditory effects of noise on health Exposure to noise disturbs sleep proportional to the amount of noise experienced in terms of an increased rate of changes in sleep stages and in number of awakenings. Habituation occurs with an increased number of sound exposures by night and across nights. One laboratory study, however, found no habituation during 14 nights of exposure to noise at maximum noise level exposure 5. Objective sleep disturbance is likely to occur if there are more than 50 noise events per night with a maximum level of 50 dBA indoors or more. In fact, there is a low association between outdoor noise levels and sleep disturbance. In the Civil Aviation Authority Study 6 around Heathrow and Gatwick airports, the relative proportion of total sleep disturbance attributable to noise increased in noisy areas but not the level of total sleep disturbance. In effect, the work suggested a symptom reporting or attribution effect rather than real noise effects. In a subsequent actigraphy study around four UK airports, sleep disturbance was studied in relation to a wide range of aircraft noise exposure over 15 consecutive nights 7. Although there was a strong association between sleep EEGs and actigram-measured awakenings and self-reported sleep disturbance, none of the aircraft noise events were associated with awakenings detected by actigram and the chance of sleep disturbance with aircraft noise exposure of Noise exposure during sleep may increase blood pressure, heart rate and finger pulse amplitude as well as body movements. There may also be after-effects during the day following disturbed sleep; perceived sleep quality, mood and performance in terms of reaction time all decreased following sleep disturbed by road traffic noise. Studies on noise abatement show that, by reducing indoor noise level, the amount of REM sleep and slow wave sleep can be increased 8. It thus seems that, although there may be some adaptation to sleep disturbance by noise, complete habituation does not occur, particularly for heart rate. Noise exposure and performance Performance may be impaired if speech is played while a subject reads and remembers verbal material, although this effect is not found with non-speech noise 10. Perceived control over and predictability of noise has been found to be important in determining effects and after-effects of noise exposure. Glass and Singer 11 found that tasks performed during noise were unimpaired but tasks that were carried out after noise had been switched off were impaired, this being reduced when subjects were given perceived control over the noise. Indeed, even anticipation of a loud noise exposure in the absence of real exposure may impair performance and an expectation of control counters this effect. Noise exposure may also slow rehearsal in memory, influence processes of selectivity in memory, and choice of strategies for carrying out tasks 1. There is also evidence that noise may reduce helping behaviour, increase aggression and reduce the processing of social cues seen as irrelevant to task performance 12. Noise and cardiovascular disease Exposure to noise causes physiological activation including increase in heart rate and blood pressure, peripheral vasoconstriction and thus increased peripheral vascular resistance. There is rapid habituation to brief noise exposure but habituation to prolonged noise is less certain 8. Occupational studies: noise and high blood pressure Many occupational studies have suggested that individuals chronically exposed to continuous noise at levels of at least 85 dB have higher blood pressure than those not exposed to noise 14, 15. In many of these studies, noise exposure has also been an indicator of exposure to other factors, both physical and psychosocial, which are also associated with high blood pressure. Unless these other risk factors are controlled, spurious associations between noise and blood pressure may arise. A recent pioneering longitudinal industrial noise study has shown that noise levels predicted raised systolic and diastolic pressure in those doing complex but not simple jobs 16, and predicts increased mortality risk. Occupational noise exposure has also recently been linked to greater risk of death from motor vehicle injury 17. One possibility is that the effects of noise on blood pressure are mediated through an intermediate psychological response such as noise annoyance 18 although this has not been convincingly proved. Intensity of noise exposure significantly associated to resting HR in women Reference Intensity of noise exposure significantly associated to resting HR in women Intensity of noise exposure significantly associated to resting HR in women Reference Intensity of noise exposure significantly associated to resting HR in women The evidence of the effects of noise on coronary risk factors has not been especially consistent: effects of noise have been shown on systolic blood pressure (but not diastolic pressure), total cholesterol, total triglycerides 20, blood viscosity, platelet count and glucose level 21. However, a recent Swedish study found that the prevalence of hypertension was higher among people exposed to time-weighted energy averaged aircraft noise levels of at least 55 dBA or maximum levels above 72 dBA around Arlanda airport, Stockholm 22. However, neither studies in coronary care units of the effect of speech noise nor studies of noise from low altitude military flights on patients on continuous cardiac monitoring have detected changes in cardiac rhythm attributable to noise 25. Endocrine responses to noise In one study, catecholamine secretion decreased when workers wore hearing protection against noise. Some studies, but not all, have shown raised cortisol in relation to noise 27. The general pattern of endocrine responses to noise is indicative of noise as a stressor, exciting short-term physiological responses, but there are inconsistencies between studies. Noise and psychiatric disorder This pathway remains unconfirmed; rather it seems that noise causes annoyance and, independently, mental ill-health also increases annoyance. A more complex model 28 incorporates the interaction between the person and their environment. In this model, the person readjusts their behaviour in noisy conditions to reduce exposure. An important addition is the inclusion of the appraisal of noise (in terms of danger, loss of environmental quality, meaning of the noise, challenges for environmental control, etc. ) and coping (the ability to alter behaviour to deal with the stressor). This model emphasizes that dealing with noise is not a passive process. Noise exposure and psychological symptoms Many of these industrial studies are difficult to interpret, however, because workers were exposed to other stressors such as physical danger and heavy work demands, in addition to excessive noise. An explicit link between aircraft noise and symptoms emerging in such studies raised the possibility of a bias towards over-reporting of symptoms 32. Notably, a study around three Swiss airports 33, which did not mention that it was related to aircraft noise, did not find any association between the level of exposure to aircraft noise and symptoms. Noise and common mental disorder In community studies such as the West London Survey of Psychiatric Morbidity 37, no overall relationship was found between aircraft noise and the prevalence of psychiatric morbidity using various indices of noise exposure. In longitudinal analyses in the Caerphilly Study, no association was found between road traffic noise and psychiatric disorder, even after adjustment for socio-demographic factors and baseline psychiatric disorder, although there was a small non-linear association of noise with increased anxiety scores 38. Overall, environmental noise seems to be linked to psychological symptoms but not to clinical psychiatric disorder. However, there may be a link to psychiatric disorder at much higher noise levels. Noise annoyance Noise is also seen as intrusive into personal privacy, while its meaning for any individual is important in determining whether that person will be annoyed by it 42. Annoyance reactions are often associated with the degree of interference that any noise causes in everyday activities, which probably precedes and leads on to annoyance 43. Overall, it seems that conversation, watching television or listening to the radio (all involving speech communication) are the activities most disturbed by aircraft noise while traffic noise, if present at night, is most disturbing for sleep. Acoustic predictors of noise annoyance in community surveys Loudness comprises the intensity of sound, the tonal distribution of sound and its duration. The evidence is mixed on the importance of both the duration and the frequency components of sound and also the number of events involved in determining annoyance 46. High frequency noise has been found to be more annoying than low frequency noise 47. Vibrations are perceived as a complement to loud noise in most community surveys of noise and are found to be important factors in determining annoyance, particularly because they are commonly experienced through other senses as well as hearing. Fields 48 found that, after controlling for noise level, noise annoyance increases with fear of danger from the noise source, sensitivity to noise, the belief that the authorities can control the noise, awareness of the non-noise impacts of the source and the belief that the noise source is not important. Combined effects of noise exposure and other stressors Stressors may act synergistically, antagonistically or not at all. Stressors may include physical, chemical, biological, social and work organizational factors 49. In a laboratory based experiment, an interaction was found between having a cold and noise exposure on simple reaction time 50. There was little difference between healthy and cold subjects’ performance tested in quiet conditions, but for subjects tested in noisy conditions (70 dBA), performance was much slower for the cold subjects.